Please wait...
About This Project
Oxidized forms of the peptide beta-amyloid (Aβ) are considered to be toxic to the brain, leading to the development of Alzheimer's disease (AD). Based on our preliminary data, we hypothesize that an immunization of an AD-model mice with a recombinant protein enriched with the oxidized amino acid methionine sulfoxide will cause the removal of toxic Aβ from brain. Consequently, this treatment is expected to inhibit the development of AD and alleviate cognitive impairment.
More Lab Notes From This Project
Browse Other Projects on Experiment
Related Projects
Dermatology AI Assistant to Empower Primary Care Providers in Zaatari Refugee Camp in Jordan
An AI-assisted dermatology tool for refugees, utilizing imaging and symptom questionnaires to improve diagnostic...
Working towards a low-cost ostomy bag that doesn't leak
Ostomy bags are a daily medical necessity for 15 million people globally. However, these bags often delaminate...
Super Slime Me: A Month of Living on an All Algae Diet
This study examines the effects of consuming only algae as a dietary source for a month. The subject, a...