156
0
0
Like?
Please wait...
About This Project
Oxidized forms of the peptide beta-amyloid (Aβ) are considered to be toxic to the brain, leading to the development of Alzheimer's disease (AD). Based on our preliminary data, we hypothesize that an immunization of an AD-model mice with a recombinant protein enriched with the oxidized amino acid methionine sulfoxide will cause the removal of toxic Aβ from brain. Consequently, this treatment is expected to inhibit the development of AD and alleviate cognitive impairment.
More Lab Notes From This Project
Browse Other Projects on Experiment
Related Projects
Developing a low-cost, rapid diagnostic for urogenital Schistosomiasis infection
We are developing a frugal diagnostic for urogenital schistosomiasis, a neglected tropical disease that...
Do fungi exposed to toxic waste produce novel antimicrobial agents?
We've discovered a few new mushrooms thriving in this harsh environment of land-disposed sewage sludge in...
Development of non-invasive diagnostics for endotheliotropic herpesvirus in free-ranging Asian elephants
Elephant endotheliotropic herpes virus hemorrhagic disease (EEHV-HD) is the leading cause of death of young...